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  Indian J Med Microbiol
 

Figure 1: Liver function of the ICR mice supplement with a Chlorella sp. (CLC) for hepatoprotective test in serum. The APAP, SM, 0.2% CLC, 0.5% CLC, and 1% CLC groups were administrated with 200 mg/kg APAP, and the normal control was administrated with PBS as placebo twice a week for 4 weeks. The Sm was fed with the chow diet containing 0.1% Silymarin (w/w). The group 0.2% CLC, 0.5% CLC, and 1% CLC were fed with the chow diet containing 0.2, 0.5, and 1% of CLC (w/w), respectively. The ICR mice were sacrificed after 4 weeks to determine the (a) GOT, (b) GPT, (c) albumin, and (d) BUN activity in serum. All of the values were expressed as the mean ± SD from 6 mice of each group. *P < 0.05, significant difference compared with control.#P < 0.05, significant difference compared with APAP-induced liver injury

Figure 1: Liver function of the ICR mice supplement with a <i>Chlorella sp</i>. (CLC) for hepatoprotective test in serum. The APAP, SM, 0.2% CLC, 0.5% CLC, and 1% CLC groups were administrated with 200 mg/kg APAP, and the normal control was administrated with PBS as placebo twice a week for 4 weeks. The Sm was fed with the chow diet containing 0.1% Silymarin (w/w). The group 0.2% CLC, 0.5% CLC, and 1% CLC were fed with the chow diet containing 0.2, 0.5, and 1% of CLC (w/w), respectively. The ICR mice were sacrificed after 4 weeks to determine the (a) GOT, (b) GPT, (c) albumin, and (d) BUN activity in serum. All of the values were expressed as the mean ± SD from 6 mice of each group. *<i>P</i> < 0.05, significant difference compared with control.<sup>#</sup><i>P</i> < 0.05, significant difference compared with APAP-induced liver injury